Diseases Treatment

What do doctors call this condition?

Multiple neuritis, peripheral neuritis, peripheral neuropathy, polyneuritis

What is this condition?

This disorder involves degeneration of peripheral nerves supplying the distal muscles of the extremities. It results in muscle weakness with sensory loss and atrophy as well as decreased or absent deep tendon reflexes. Although peripheral nerve degeneration can occur at any age, incidence is highest in men between ages 30 and 50. Because onset is usually subtle, a person may compensate by overusing unaffected muscles; however, the disease may begin rapidly with severe infection and chronic alcohol intoxication. If the cause can be identified and eliminated, the prognosis is good.

What causes it?

Causes of peripheral nerve degeneration include:

• chronic intoxication (with ethyl alcohol, arsenic, lead, carbon disulfide, benzene, phosphorus, and sulfonamides)

• infectious diseases (meningitis, diphtheria, syphilis, tuberculosis, pneumonia, and mumps)

• metabolic and inflammatory disorders (gout, diabetes, rheumatoid arthritis, polyarteritis nodosa, and lupus)

• nutritional disorders (beriberi and other vitamin deficiencies, and malnourishment) .

What are its symptoms?

The signs and symptoms of peripheral nerve degeneration develop slowly, and the disease usually affects the motor and sensory nerve fibers. The condition typically produces flaccid paralysis, wasting, loss of reflexes, pain of varying intensity, loss of ability to perceive vibratory sensations, and numbness, tingling, increased sensitivity to pain or touch, or anesthesia in the hands and feet. Deep-tendon reflexes are diminished or absent, and atrophied muscles are tender or hypersensitive to pressure or palpation. Footdrop may also be present. Skin manifestations include glossy red skin and decreased sweating. Many people with the disease have a history of clumsiness and complain of frequent vague sensations.

How is it diagnosed?

The person’s history and physical exam reveal characteristic distribution of motor and sensory deficits. Electromyography may show a delayed action potential if peripheral nerve degeneration impairs motor nerve function.

How is it treated?

Effective treatment consists of supportive measures to relieve pain, adequate bed rest, and physical therapy, as needed. Most important, however, the underlying cause must be identified and corrected. For instance, it’s essential to identify and remove the toxic agent, correct nutritional and vitamin deficiencies (the person needs a high-calorie diet rich in vitamins, especially the B-complex group), or counsel the person to avoid alcohol.
What can a person with peripheral nerve degeneration do?

• Rest frequently and avoid using the affected arm or leg. To avoid bed sores, use a foot cradle. To prevent contractures, use splints, boards, braces, or other orthopedic appliances

• After the pain subsides, passive range-of-motion exercises or massage may be beneficial. The doctor may recommend electrotherapy for nerve and muscle stimulation.

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What is this Condition?

Gout is a metabolic disease marked by localized deposits of uric acid salts that are normally excreted through the kidneys. The deposits cause painfully arthritic joints. Gout usually strikes the feet and legs of men over age 30 and women past menopause. In elderly people, it is linked to other diseases.

Though gout may disappear for years between attacks, it can lead to disability or crippling. Fortunately, most people get better with treatment.

What Causes Gout?

Although the exact cause is unknown, gout seems linked to a genetic defect in metabolism, which causes overproduction and retention of uric acid. Too much uric acid leads to urate deposits in the joints or tissues, causing local damage. Secondary gout, linked to other conditions (such as obesity, diabetes, high blood pressure, sickle cell anemia, and kidney disease) or to drug therapy, produces similar harmful substances.

Another condition, called pseudogout, or chondrocalcinosis, causes arthritic pain too, but for different reasons.

What are its Symptoms?

Gout develops in four stages (asymptomatic, acute, intercritical, and chronic) that produce the following findings:

• In asymptomatic gout, urate levels rise in the blood but produce no symptoms. Later, gout may cause high blood pressure or show up in severe back pain.

• The first acute attack strikes suddenly and peaks quickly, causing extreme pain in one or only a few joints. Affected joints feel hot, tender, inflamed, and look dusky red or bruised. The joint of the big toe usually becomes inflamed first, then the instep, ankle, heel, knee, or wrist joints. Some attacks pass quickly and then come back at irregular intervals. Severe attacks may last for days or weeks.

• Intercritical periods are the symptom-free intervals between gout attacks. Most people have a second attack within 6 months to 2 years, but others are symptom-free for 5 to 10 years. Those delayed attacks can strike untreated people with longer-lasting, severe pain in several joints, sometimes all at once and sometimes in one joint after another.

• Eventually, chronic gaur sets in. This final, continuous stage shows up in persistently painful joints, with large urate deposits in the cartilage, membranes between the bones, tendons, and soft tissue. Deposits form primarily in arms and legs and, rarely, in organs, such as the kidneys and heart lining.

The skin over the deposits may develop sores and release a chalky, white material or pus. Chronic inflammation and urate deposition progress to further restrict movement and harm the person’s general health, possibly including formation of kidney stones.

How is it diagnosed?

The doctor can find evidence of gout in fluid taken from an inflamed joint or a deposit and by checking the level of uric acid in the blood. In chronic gout, X-rays show damage to the cartilage and bones.

How is it Treated?

The doctor first tries to stop the pain and prevent complications by suggesting bed rest and protection for the painful joints. Hot or cold packs and pain relievers may help with mild attacks. For more severe attacks and chronic gour, treatment may include the following:

• drugs to reduce inflammation, including Colsalide, Butazolidin, and Indocin, and injections of corticosteroids or corticotropin

• slower-acting drugs to reduce the uric acid level in the blood, including Zyloprim, Colsalide, Benemid, and Anturane

• diet changes, primarily to avoid alcohol and some rich foods; obese people should try to lose weight because the extra weight puts more stress on painful joints.

• surgery to improve joint function or correct deformities. Deposits must be opened and drained if they become infected or ulcerated. Deposits can also be cut out to prevent ulceration, improve the joint’s appearance, or make it easier to wear shoes or gloves.

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